postsynaptic membrane

The presynaptic and postsynaptic membrane were damaged so seriously that the typical synaptic structure did not exist.

突触前后膜被破坏，典型的突触结构已不存在。

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Study on Function Change of AMPA Receptor on Postsynaptic Membrane after Ischemic Insult and Related Molecular Mechanism

缺血损伤后突触后膜AMPA受体的功能变化及分子机制研究

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At 12 weeks after implantation, many synaptophysins gathered on the surface of denervated skeletal muscles and the form of postsynaptic membrane keep well and was similar as the one of normal membrane.

细胞移植后12周，实验组失神经肌肉表面突触素大量聚集，突触后膜形态较好，已接近正常肌肉突触后膜的形态。

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Objective To explore the changes of glutamate receptor ( GluR) 1~ 3 in the postsynaptic membrane and the effects on the delayed neuron death after oxygen-glucose deprivation ( OGD).

目的探时OGD损伤后神经元突触后膜GluR1～3含量变化及其在神经元延迟性细胞死亡中的作用。

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Hereafter postsynaptic membrane potential was depolarized gradually, and the maximum depolarization value was 27. 0 mV. The frequency of mEPP increased markedly after 70. 3 min MTX administered, 32 folds compared with control.

随后，突触后膜开始逐渐去极化，最大去极化27.0mV。62.7min后小终板电位（mEPP）频率逐渐增高，到70.3min达到最高频率，比给药前增加了32倍。

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The changes in the thickness of postsynaptic membrane during acupuncture analgesia

电针镇痛时突触后膜厚度的变化

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Synapses are connections between neurons, biological signals are transferred from the presynaptic membrane by the synaptic transmission to the postsynaptic membrane.

突触是神经元之间的连接部位，生物信号从突触前膜经突触传递到突触后膜。

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And the opening proportion of chemical-gated ion channels on postsynaptic membrane was positively related to the potential of presynaptic membrane.

突触后膜上化学门控离子通道的开放比例与突触前膜电位呈正相关；

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Glutamate binds to receptors on the postsynaptic neuronal membrane to activate the influx of sodium and calcium.

GLU与突触后神经元膜上的受体相结合，导致钠离子和钙离子内流，引起一系列的生物化学级联反应。

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Wet weight of triceps surae was measured. The denervated muscle was given HE staining and Mallory staining, while the frozen muscle biopsy tissue was given Immunofluorescence with postsynaptic membrane AchR specific antibody.

测量小腿三头肌肌湿重，并分别应用HE染色、Mallory三色染色的方法对失神经肌肉组织进行染色，同时应用突触后膜乙酰胆碱受体（AchR）特异性抗体对肌肉冰冻切片进行免疫荧光染色。

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Changes of GluRs on postsynaptic membrane and delayed neuron death after OGD

OGD后突触后膜GluRs含量变化及神经元延迟性死亡研究

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The postsynaptic membrane of S-, T-and G-types is apparently thicker than the presynaptic membrane and showed to be asymmetrical.

S型、T型和G型突触后膜均比前膜明显增厚，呈不对称性。

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Presynaptic membrane, postsynaptic membrane and synaptic cleft were clear. Presynaptic terminals had more round clearest vesicles, and postsynaptic membrane has a high density electron. 3.

突触前膜、突触后膜和突触间隙清晰，突触前终末有较多清亮的圆形囊泡，突触后膜有电子密度高的致密物质。

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Muscle-specific kinase ( MuSK) is a transmembrane protein which is related with acetylcholine receptor, in the development of skeletal muscle, MuSK is a primary ingredient to establish the main synaptic skeleton of the postsynaptic membrane formation.

肌肉特异性激酶（muscle-specific kinase，MuSK）是一种与乙酰胆碱受体相关的跨膜蛋白，在骨骼肌的发展中，MuSK是建立突触后膜时组建主要突触骨架的首要成分。

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Neurotransmitter receptors mediate signal transduction postsynaptic membrane of synaptic connections between neurons in the nervous system.

大脑中神经元突触间的信号传递是由许多神经递质受体介导的.

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A current prevalent model regarding postsynaptic development is that a group of key scaffolding proteins orchestrate the assembly of macromolecular complex at the postsynaptic membrane by providing multiple protein docking sites as well as connections to the actin cytoskeleton.

目前比较流行的突触发育的模式是一群主要的支架蛋白通过提供多个锚定位点和与肌动蛋白细胞骨架连接位点，将大分子复合物装配在突触后膜。

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Submicroscopic structure of postsynaptic membrane and subsynaptic dense body

突触后膜和突触下致密小体的亚显微结构

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A significant reduction in the number of synaptic with nuclear presynaptic membrane and postsynaptic membrane, and a low density in postsynaptic dense zone.

模型组神经元突触数量明显减少，突触前、后膜结构模糊不清，突触后致密区密度低。

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Neurotransmitter receptors mediate signal transduction at the postsynaptic membrane of synaptic connections between neurons in the nervous system.

大脑中神经元突触间的信号传递是由许多神经递质受体介导的。

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Results The length of postsynaptic membrane decreased obviously. The ratio of the length of postsynaptic with presynaptic membrane, and that the motor nerve terminal area with postsynaptic membrane area diminished. The AChR on the postsynaptic membrane decreased by 35.9 percent as well.

结果MG时突触后膜长度缩短，后、前膜长度比值变小，神经末端面积及其与突触后膜面积之比变小，突触后膜上的乙酰胆碱受体（AChR）减少了35.9%。

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